References

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Romandini M, Soldini MC, Montero E, Sanz M. Epidemiology of mid-buccal gingival recessions in NHANES according to the 2018 World Workshop Classification System. J Clin Periodontol. 2020; 47:1180-1190 https://doi.org/10.1111/jcpe.13353
Axéll T, Koch G. Traumatic ulcerative gingival lesion. J Clin Periodontol. 1982; 9:178-183 https://doi.org/10.1111/j.1600-051x.1982.tb02057.x
Beck JD, Koch GG, Offenbacher S. Attachment loss trends over 3 years in community-dwelling older adults. J Periodontol. 1994; 65:737-743 https://doi.org/10.1902/jop.1994.65.8.737
Pires IL, Cota LO, Oliveira AC Association between periodontal condition and use of tongue piercing: a case-control study. J Clin Periodontol. 2010; 37:712-718 https://doi.org/10.1111/j.1600-051X.2010.01584.x
Kapferer I, Benesch T, Gregoric N Lip piercing: prevalence of associated gingival recession and contributing factors. A cross-sectional study. J Periodontal Res. 2007; 42:177-183 https://doi.org/10.1111/j.1600-0765.2006.00931.x
Ainamo J, Löe H. Anatomical characteristics of gingiva. A clinical and microscopic study of the free and attached gingiva. J Periodontol. 1966; 37:5-13 https://doi.org/10.1902/jop.1966.37.1.5
Friedman N. Mucogingival surgery: the apically repositioned flap. J Periodontol. 1962; 33:328-340
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Dorfman HS, Kennedy JE, Bird WC. Longitudinal evaluation of free autogenous gingival grafts. J Clin Periodontol. 1980; 7:316-324 https://doi.org/10.1111/j.1600-051x.1980.tb01974.x
Kennedy JE, Bird WC, Palcanis KG, Dorfman HS. A longitudinal evaluation of varying widths of attached gingiva. J Clin Periodontol. 1985; 12:667-675 https://doi.org/10.1111/j.1600-051x.1985.tb00938.x
Chambrone L, Tatakis DN. Long-term outcomes of untreated buccal gingival recessions: a systematic review and meta-analysis. J Periodontol. 2016; 87:796-808 https://doi.org/10.1902/jop.2016.150625
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Sullivan HC, Atkins JH. Free autogenous gingival grafts. I. Principles of successful grafting. Periodontics. 1968; 6:121-129
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Abolfazli N, Saber FS, Lafzi A, Eskandari A. Evaluation of alteration in mucogingival line location following use of subepithelial connective tissue graft. Indian J Dent Res. 2010; 21:174-178 https://doi.org/10.4103/0970-9290.66628
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The free gingival graft

From Volume 50, Issue 1, January 2023 | Pages 34-39

Authors

Alaa Guni

Specialty Registrar in Periodontology, Guy‘s Hospital, London

Articles by Alaa Guni

Email Alaa Guni

Luigi Nibali

Professor/Honorary Consultant in Periodontology; Centre for Host-Microbiome Interactions, Faculty of Dentistry, Oral and Craniofacial Sciences, King's College London

Articles by Luigi Nibali

Abstract

Recession defects are widely prevalent in the adult population, with some patients being impacted by complications, including the difficulty of cleaning such sites and the risk of continued recession progression. The free gingival graft (FGG) is a form of mucogingival surgery that attempts to improve the height of the keratinized tissue (KT), as well as augment the thickness of the gingival phenotype to facilitate meticulous oral hygiene. This article discusses the indications for the surgery, the procedure, including post-operative care, and possible modifications of the technique.

CPD/Clinical Relevance: Professional intervention involving a free gingival graft may be the only way to empower patients to clean the recession site without discomfort.

Article

Gingival recession can be defined as the exposure of the root surface due to apical migration of the gingival margin.1 Such defects can be localized or generalized, being present interproximally and/or labially/lingually. The latest NHANES data suggest that recession sites are widely prevalent, with 91.6% of the adult population presenting with the condition. Presence of gingival recession is associated with older age, female gender, European ethnicity and the mandibular arch.2

Recession can have a multifactorial aetiology and, therefore, a careful history and examination are required, as well as appropriate investigation of the affected sites (Table 1). The mechanism of recession is not fully understood, but the presence of persistent inflammation is required to lead to recession. Such inflammation can lead to the breakdown of the underlying connective tissue, which eventually leads to gingival tissue recession. Causes of inflammation can be mechanical, such as overzealous brushing3 or the use of overly hard bristles. Traumatic occlusion can also lead to recession in cases of a deep overbite in incisal Class 2 or 3 relationships where the incisal edges can directly contact the soft gingival tissues. Inflammation can also be attributed to a bacterial aetiology, where persistent plaque presence can lead to an inflammatory host response and, if left untreated, may develop into periodontitis.4 Tongue studs and lip piercings are also associated with recession. Individuals with tongue studs can have up to an 11-times greater chance of lingual recession of the mandibular incisors compared to non-users.5 Meanwhile, lip piercings that are positioned close to the cemento-enamel junction (CEJ) are associated with buccal/labial recessions.6 Both objects increase the risk of recession with long-term use.

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